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The effects of chronic ethanol administration on the hypothalamic–pituitary-adrenal axis and catecholamine activity in the Fischer 344 rat.
The effects of chronic ethanol consumption on the hypothalamic-pituitary-adrenal (HPA) axis and catecholamine activity were assessed using male Fischer 344 rats. Following a rapid daily dose of ethanol (10 g/kg body weight), plasma corticosterone (CS) levels rose from undetectable to peak at day 3 and decline by day 10. The daily-dose-induced plasma CS response was characterized by a significant increase in the CS concentration on days 3, 5, and 10. Adrenal cortisol enzyme activities and turnover rates were not significantly different from controls. Acute ethanol administration (4 g/kg body weight) increased adrenal CS levels from control baseline values, and was followed by a rapid decline in CS levels to values not significantly different from controls by 8 h. These observations indicate that the changes in plasma CS levels are not mediated by acute ethanol effects on the HPA axis. Serum and adrenal catecholamines (CA) levels were not significantly altered by chronic ethanol administration. In addition, chronic ethanol administration had no effect on the sensitivity of the adrenal CA system to stimulation by acute ethanol exposure. These data suggest that the decrease in the CS response to acute ethanol administration is probably not due to effects on the HPA axis and/or alterations in adrenal CA system sensitivity.1 of 6 View Caption

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